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InicioE-BooksE-Books Médicos The Resolution of Inflammation – Apoptosis
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World of Microbiology and Immunology - 2 Volume Set

The Resolution of Inflammation – Apoptosis

Product details

  • Series: Progress in Inflammation Research
  • Hardcover: 240 pages
  • Publisher: Birkhäuser; 2008 edition (February 6, 2008)
  • Language: English
  • ISBN-10: 3764375051
  • ISBN-13: 978-3764375058
  • Product Dimensions: 6.7 x 0.6 x 9.6 inches
  • Shipping Weight: 1.5 pounds

This book provides readers with an up-to-date and comprehensive view on the resolution of inflammation and on new developments in this area, including pro-resolution mediators, apoptosis, macrophage clearance of apoptotic cells, possible novel drug developments.

Much progress has been made in elucidating the mechanisms regulating the induction and progression of inflammatory diseases. More recently, however, it has become apparent that the manipulation of mechanisms governing the resolution of inflammation is an important way to develop novel strategies for the therapy of such diseases. This book provides an up-to-date and comprehensive view on the resolution of inflammation and on new developments in this area (e.g. pro-resolution mediators, apoptosis, macrophage clearance of apoptotic cells, possible novel drug developments). The chapters are written by leading scientists in the field.

This volume contains major contributions from an international panel of experts who describe the basic processes regulating the resolution of inflammation including apoptosis, macrophage clearance of apoptotic cells and novel pro-resolution lipid mediators. In addition, there are sections that describe how existing anti-inflammatory drugs such as aspirin and glucocorticoids may influence these resolution processes. There are three chapters devoted to describing fine examples of clinically relevant inflammatory disease areas where much progress has been made in understanding resolution. We feel that we are at the beginning of a rapidly burgeoning and exciting area of inflammation research where new advances are being made in understanding the resolution of inflammation.

This topical volume is of interest to medical and scientific researchers in academia and industry working on inflammatory and cardiovascular diseases such as arthritis, asthma, bronchitis, atherosclerosis, and more.

Categoría: E-Books Médicos
  • Descripción
Descripción

It was with tremendous enthusiasm that we endeavoured to compile and edit this volume for Progress in Inflammation Research describing novel findings and developments pertaining to the processes governing the resolution of inflammation. It is perhaps surprising that this topic had, to our knowledge, not previously been covered as a separate subject area in a dedicated monograph given what now seems such an obvious thing to do. Historically, researchers have focussed and have made great advances on the initiation and propagation of inflammation. Little attention had been specifically devoted to elucidating the mechanisms orchestrating the resolution of inflammation, although a variety of mechanisms that limit the inflammatory response had been described (e.g., mediator dissipation and deactivation; exogenous mediator removal or reduction; receptor, cell and tissue desensitisation to mediators; identification of agents with anti-inflammatory potential such as IL-10, IL-1 receptor antagonists, TGF-􀁠, etc).

It is now believed that manipulation of more recently described processes, recognised as being actively involved in resolution, are therapeutically manipulatable for the treatment of inflammatory diseases. Indeed, patients with chronic inflammatory diseases are by necessity treated in order to reduce established and persistent inflammation with the added hope of preventing further progression of the inflammatory response. It has recently become evident that many of the anti-inflammatory agents currently used in the clinical setting influence inflammatory resolution. For example, glucocorticoids have been shown to influence processes now recognised as being important mechanisms allowing resolution to occur; namely glucocorticoids trigger apoptosis (programmed cell death) in most leukocytes (the neutrophil however is a notable exception) and augment apoptotic cell clearance by phagocytes. Similarly, aspirin, the most widely used NSAID, is involved in an unorthodox biosynthetic pathway yielding important lipid mediators (e.g., 15-epi-lipoxin A4 and 15-epi-lipoxin B4) actively involved in the resolution process.

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